prof. Jody Haigh
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Non-angiogenic functions of VEGF
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- Journal Article
- A1
- open access
Vascular endothelial growth factor directly inhibits primitive neural stem cell survival but promotes definitive neural stem cell survival
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Direct evidence for endothelial vascular endothelial growth factor receptor-1 function in nitric oxide-mediated angiogenesis
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Vascular endothelial growth factor A signaling in the podocyte-endothelial compartment is required for mesangial cell migration and survival
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- Journal Article
- A1
- open access
Conditional and inducible transgene expression in mice through the combinatorial use of Cre-mediated recombination and tetracycline induction
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Aberrant bone formation in mice over-expressing specific VEGF isoforms in cartilage.
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Activated Fps/Fes partially rescues the in vivo developmental potential of Flk1-deficient vascular progenitor cells
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Cre recombinase specificity defined by the tau locus
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Loss of vascular endothelial growth factor A activity in murine epidermal keratinocytes delays wound healing and inhibits tumor formation
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Impaired intervertebral disc formation in the absence of Jun
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- Journal Article
- A1
- open access
Glomerular-specific alterations of VEGF-A expression lead to distinct congenital and acquired renal diseases
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Cortical and retinal defects caused by dosage-dependent reductions in VEGF-A paracrine signaling
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Hyperglycemia-induced vasculopathy in the murine conceptus is mediated via reductions of VEGF-A expression and VEGF receptor activation
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Conditional inactivation of VEGF-A in areas of collagen2a1 expression results in embryonic lethality in the heterozygous state
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The fps/fes tyrosine kinase is expressed in myeloid, vascular endothelial, epithelial, and neuronal cells and is localized in the trans-Golgi network
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The Fps/Fes protein-tyrosine kinase promotes angiogenesis in transgenic mice