Show
Sort by
-
Melanoma inhibitory activity, a biomarker related to chondrocyte anabolism, is reversibly suppressed by proinflammatory cytokines in rheumatoid arthritis
-
Anti-TNFalpha induced apoptosis contributes to the induction of autoantibodies but not the decrease of synovial inflammation in spondyloarthropathy (SpA)
-
Non-pathogenic antinuclear antibodies contribute to the clearance of apoptotic antigens released during TNF alpha blockade in spondyloarthritis (SpA)
-
Endogenous HLA-DR-restricted presentation of the cartilage antigens human cartilage gp-39 and melanoma inhibitory activity in the inflamed rheumatoid joint
-
A randomized controlled trial with an anti-CCL2 (anti-monocyte chemotactic protein 1) monoclonal antibody in patients with rheumatoid arthritis
-
Synovial inflammation does not change in the absence of effective treatment: implications for the use of synovial histopathology as biomarker in early phase clinical trials in rheumatoid arthritis
-
Identification of synovial biomarkers of response to experimental treatment in early-phase clinical trials in spondylarthritis
-
Anti-TNF alpha induced apoptosis contributes to the induction of Autoantibodies but not the decrease of synovial inflammation in spondyloarthropathy (SPA)
-
Growth promoting effect of human cartilage glycoprotein-39 (HC gp-39) on synovial fibroblasts
-
Anti-TNFalpha induced apoptosis contributes to the induction of autoantibodies but not the decrease of synovial inflammation in spondyloarthropathy
-
Histological evidence that infliximab treatment leads to downregulation of inflammation and tissue remodelling of the synovial membrane in spondyloarthropathy
-
Differential expression and response to anti-TNF alpha treatment of infiltrating versus resident tissue macrophage subsets in autoimmune arthritis
-
Fibrin deimination in synovial tissue is not specific for rheumatoid arthritis but commonly occurs during synovitides
-
Anti-citrullinated peptide antibodies may occur in patients with psoriatic arthritis
-
IgM anti-dsDNA antibodies are the main anti-nuclear reactivity induced by infliximab but not etanercept treatment: biological and clinical implications
-
Synovial intracellular citrullinated proteins colocalizing with peptidyl arginine deiminase are pathophysiologically relevant antigenic determinants of rheumatoid arthritis-specific humoral autoimmunity
-
Increased systemic and local expression of toll-like receptor 2 and 4 in spondyloarthropathy is downmodulated by tumour necrosis factor alpha blockade
-
Infliximab, but not etanercept, induces IgM anti-double-stranded DNA autoantibodies as main antinuclear reactivity : biologic and clinical implications in autoimmune arthritis
-
Impact of natural changes in disease activity on synovial histopathology in rheumatoid arthritis
-
Tumor necrosis factor a blockade treatment down-modulates the increased systemic and local expression of toll-like receptor 2 and toll-like receptor 4 in spondylarthropathy