Marjory Desmedt
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Interleukin-15 redirects the outcome of a tolerizing T-cell stimulus from apoptosis to anergy.
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Macrophages present pinocytosed exogenous antigen via MHC class I whereas antigen ingested by receptor-mediated endocytosis is presented via MHC class II.
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Tumoral environment triggers transcript anomalies in established tumors : induction of altered gene expression and of aberrant, truncated and B2 repeat-containing gene transcripts
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Lipopolysaccharide regulates macrophage fluid phase pinocytosis via CD14-dependent and CD14-independent pathways.
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Cathepsin B-mediated activation of the proinflammatory caspase-11
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Macrophages induce cellular immunity by activating Th1 cell responses and suppressing Th2 cell responses.
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Differentiation of EL4 lymphoma cells by tumoral environment is associated with inappropriate expression of the large chondroitin sulfate proteoglycan PG-M and the tumor-associated antigen HTgp-175.
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Quiescence-inducing and antiapoptotic activities of IL-15 enhance secondary CD4(+) T cell responsiveness to antigen.