dr. Karen Heyninck
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Identificatie en karakterisering van cellulaire inhibitoren van NF-kB-afhankelijke genexpressie
(1999) -
The zinc finger protein A20 interacts with a novel anti-apoptotic protein which is cleaved by specific caspases.
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The cytokine-inducible zinc finger protein A20 inhibits IL-1-induced NF-kappa B activation at the level of TRAF6.
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Inhibition of Tumor Necrosis Factor-induced necrotic cell death by the zinc finger protein A20.
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- Journal Article
- A1
- open access
The zinc finger protein A20 inhibits TNF-induced NF-kappa B-dependent gene expression by interfering with an RIP- or TRAF2-mediated transactivation signal and directly binds to a novel NF-kappa B-inhibiting protein ABIN.
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Tumour necrosis factor: intracellular mechanism of action
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A20 inhibits NF-kappa B activation independently of binding to 14-3-3 proteins.
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A20, an inhibitor of cell death, self-associates by its zinc finger domain.
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TNF-induced intracellular signaling leading to gene induction or to cytotoxicity by necrosis or by apoptosis.
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ENHANCEMENT OF TUMOR-NECROSIS-FACTOR CYTOTOXICITY BY LITHIUM-CHLORIDE IS ASSOCIATED WITH INCREASED INOSITOL PHOSPHATE ACCUMULATION.