Wim Van Molle
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Protection against TNF-induced lethal shock by soluble guanylate cyclase inhibition requires functional inducible nitric oxide synthase.
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- Journal Article
- A1
- open access
The major acute-phase protein, serum amyloid P component, in mice is not involved in endogenous resistance against tumor necrosis factor alpha-induced lethal hepatitis, shock, and skin necrosis.
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Onderzoek naar de mechanismen leidend tot Tumor Necrosis Factor-geïnduceerde leverschade en hypotensie
(1999) -
The role of complement activation in tumour necrosis factor-induced lethal hepatitis.
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Caspase-1 is not involved in experimental hepatitis in mouse.
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Activation of caspases in lethal experimental hepatitis and prevention by acute phase proteins.
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al-Acid glycoprotein and al-antitrypsin inhibit TNF-induced but not anti-Fas-induced apoptosis of hepatocytes in mice
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al-Antitrypsin inhibits the lethal response to TNF in mice
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Optimalization and application in TNF-related models of a method to estimate NO production in vivo.