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Is chronic exposure of the blood-brain barrier to relatively low L-β-ODAP concentrations a possible entry point of L-β-ODAP into the central nervous system?
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- PhD Thesis
- open access
Study of the etiology of neurolathyrism, a human neurodegenerative disease with nutritional causes
(2011) -
- Journal Article
- A4
- open access
On the molecular mechanisms of konzo and neurolathyrism
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L-β-N-oxalyl-α,β-diaminopropionic acid toxicity in motor neurons
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Studies on neurolathyrism in Ethiopia: dietary habits, perception of risk and prevention
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Unraveling the mechanism of β-N-oxalyl-α,β-diaminopropionic acid (β-ODAP) induced excitotoxicity and oxidative stress, relevance for neurolathyrism prevention
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L-β-ODAP alters mitochondrial Ca2+ handling as an early event in excitotoxicity
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Hemichannels contribute to Ca2+ dynamics in smooth muscle cells in acutely isolated small mesenteric arteries
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Neuro- and gliotoxicity of the excitatory amino acid beta-ODAP involves impaired free radical defence
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Unraveling the mechanism of beta-ODAP induced excitotoxicity and oxidative stress, relevance for neurolathyrism prevention