- ORCID iD
- 0000-0001-8823-0000
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- Journal Article
- A1
- open access
Targeting gliovascular connexins prevents inflammatory blood-brain barrier leakage and astrogliosis
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- Journal Article
- A1
- open access
Cx43 channels and signaling via IP3/Ca2+, ATP, and ROS/NO propagate radiation-induced DNA damage to non-irradiated brain microvascular endothelial cells
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- Journal Article
- A1
- open access
Targeting MAPK phosphorylation of Connexin43 provides neuroprotection in stroke
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Calcium, a pivotal player in photodynamic therapy?
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Calcium, oxidative stress and connexin channels, a harmonious orchestra directing the response to radiotherapy treatment?
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Connexin channels at the glio-vascular interface : gatekeepers of the brain
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At the cross-point of connexins, calcium, and ATP : blocking hemichannels inhibits vasoconstriction of rat small mesenteric arteries
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Pannexin1 as mediator of inflammation and cell death
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Electroporation loading and dye transfer : a safe and robust method to probe gap junctional coupling
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Into rather unexplored terrain : transcellular transport across the blood-brain barrier
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Electroporation loading of membrane-impermeable molecules to investigate intra- and intercellular Ca2+ signaling
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Flash photolysis of caged IP3 to trigger intercellular Ca2+ waves
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Electroporation loading and flash photolysis to investigate intra- and intercellular Ca2+ signaling
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- Journal Article
- A1
- open access
Intracellular cleavage of the Cx43 C-terminal domain by matrix-metalloproteases : a novel contributor to inflammation?
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Connexin and pannexin signaling pathways, an architectural blueprint for CNS physiology and pathology?
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- Journal Article
- A1
- open access
Fluoxetine suppresses calcium signaling in human T lymphocytes through depletion of intracellular calcium stores
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- Journal Article
- A1
- open access
The connexin43 mimetic peptide Gap19 inhibits hemichannels without altering gap junctional communication in astrocytes
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The dual face of connexin-based astroglial Ca2+ communication: a key player in brain physiology and a prime target in pathology
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A new angle on blood–CNS interfaces: a role for connexins?
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- Journal Article
- A1
- open access
Neurological manifestations of oculodentodigital dysplasia: a Cx43 channelopathy of the central nervous system?
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Peptides and peptide-derived molecules targeting the intracellular domains of Cx43: gap junctions versus hemichannels
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Endothelial calcium dynamics, connexin channels and blood-brain barrier function
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Connexin targeting peptides as inhibitors of voltage- and intracellular Ca2+-triggered Cx43 hemichannel opening
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Inhibiting connexin channels protects against cryopreservation-induced cell death in human blood vessels
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IP₃, a small molecule with a powerful message
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Selective inhibition of Cx43 hemichannels by Gap19 and its impact on myocardial ischemia/reperfusion injury
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Connexin mimetic peptides inhibit Cx43 hemichannel opening triggered by voltage and intracellular Ca2+ elevation
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Is chronic exposure of the blood-brain barrier to relatively low L-β-ODAP concentrations a possible entry point of L-β-ODAP into the central nervous system?
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Paracrine signaling through plasma membrane hemichannels
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Low extracellular Ca2+ conditions induce an increase in brain endothelial permeability that involves intercellular Ca2+ waves
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Connexin channels provide a target to manipulate calcium dynamics and blood brain barrier permeability
(2012) -
Transfer of IP3 through gap junctions is critical, but not sufficient, for the spread of apoptosis
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Connexin 43 hemichannels contribute to cytoplasmic Ca2+ oscillations by providing a bimodal Ca2+-dependent Ca2+ entry pathway
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Memichannels contribute to Ca2+ dynamics and contractility of smooth muscle cells in acutely isolated small mesenteric arteries
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Connexin channels provide a target to manipulate brain endothelial calcium dynamics and blood brain barrier permeability
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L-β-N-oxalyl-α,β-diaminopropionic acid toxicity in motor neurons
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Pannexin channels in ATP release and beyond: an unexpected rendezvous at the endoplasmic reticulum
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- Conference Paper
- C3
- open access
Hemichannel involvement in CA2+ dynamics and contractility of smooth muscle cells in acutely isolated small mesenteric arteries
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L-β-ODAP alters mitochondrial Ca2+ handling as an early event in excitotoxicity
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Connexin32 hemichannels contribute to the apoptotic-to-necrotic transition during Fas-mediated hepatocyte cell death
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Hemichannels contribute to Ca2+ dynamics in smooth muscle cells in acutely isolated small mesenteric arteries
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Neuro- and gliotoxicity of the excitatory amino acid beta-ODAP involves impaired free radical defence
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Neuro- and gliotoxicity of the excitatory amino acid beta-ODAP involves impaired free radical difference
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Involvement of connexin hemichannels in dynamic calcium changes and permeability of blood-brain barrier endothelial cells
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Cell-cell communication of apoptosis via connexin channels in a C6 glioma cell model.
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Inhibition of connexin 43 hemichannel responses with high cytoplasmic calcium concentrations is mediated by mechanisms different from calcium activation of hemichannel responses
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Calcium regulation of connexin-43 hemichannel-mediated ATP release in glial cells
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Cell death communication via connexin channels investigated in a C6 glioma cell model
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Ca2+ regulation of connexin 43 hemichannels in C6 glioma and glial cells
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Connexin 43 hemichannels contribute to the propagation of apoptotic cell death in a rat C6 glioma cell model.
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Gliotransmitter release via Connexin 43 hemichannels: role of cytoplasmic changes
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Cx43 hemichannel regulation by intracellular calcium: involvement of different signaling pathways
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Inhibition of Cx43 hemichannel responses with High Ca2+i is mediated by mechanisms different from calcium activation of hemichannel responses
(2008) 194. -
In situ bipolar Electroporation for localized cell loading with reporter dyes and investigating gap junctional coupling
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Connexin channels mediate propagation of apoptosis in a C6 glioma cell model
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Neurobarrier coupling in the brain: Adjusting glucose entry with demand
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Connexin hemichannels and gap junction channels are differentially influenced by lipopolysaccharide and basic fibroblast growth factor
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Connexin channels, possible mediators in apoptosis spread in a glioma cell model