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The autoinflammation-associated NLRC4<SUP>V341A</SUP> mutation increases microbiota-independent IL-18 production but does not recapitulate human autoinflammatory symptoms in mice
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GSDMD drives canonical inflammasome-induced neutrophil pyroptosis and is dispensable for NETosis
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MCC950/CRID3 potently targets the NACHT domain of wild-type NLRP3 but not disease-associated mutants for inflammasome inhibition
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Caspase-1 engagement and TLR-induced c-FLIP expression suppress ASC/caspase-8-dependent apoptosis by inflammasome sensors NLRP1b and NLRC4
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Neutrophilic NLRP3 inflammasome-dependent IL-1β secretion regulates the γδT17 cell response in respiratory bacterial infections
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Familial Mediterranean fever mutations lift the obligatory requirement for microtubules in Pyrin inflammasome activation
(2016) PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA. 113(50). p.14384-14389 -
Complementarity and redundancy of IL-22-producing innate lymphoid cells
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Does caspase-12 suppress inflammasome activation?
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Escherichia coli α-hemolysin counteracts the anti-virulence innate immune response triggered by the Rho GTPase activating toxin CNF1 during bacteremia
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Protective and detrimental roles of inflammasomes in disease
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Nlrp6 promotes recovery after peripheral nerve injury independently of inflammasomes
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Non-classical ProIL-1beta activation during mammary gland infection is pathogen-dependent but caspase-1 independent
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Optimalization and characterization of a novel intraductal mouse mammary tumor model
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Adaptation of an intraductal mouse mastitis model to study the effect of inflammation in breast cancer metastasis
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Caspase-11 is expressed in the colonic mucosa and protects against dextran sodium sulfate-induced colitis
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Sensing the enemy within: how macrophages detect intracellular Gram-negative bacteria
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Non-classical ProIL-1beta activation during mammary gland infection is pathogen-dependent but caspase-1 independent
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Simultaneous targeting of IL-1 and IL-18 is required for protection against inflammatory and septic shock
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Escherichia coli induces bovine neutrophil cell death independent from caspase-3/-7/-1, but with phosphatidylserine exposure prior to membrane rupture
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An inactivating caspase-11 passenger mutation muddles sepsis research