Sandrine Herbelet
work address
De Pintelaan 185
9000 Gent
9000 Gent
ORCID iD
0000-0003-1911-01731 – 10 of 16
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- 2016
- Activation of osmolyte pathways in inflammatory myopathy and Duchenne muscular dystrophy points to osmoregulation as a contributing pathogenic mechanism
- NFAT5 in Duchenne muscular dystrophy cultured myoblasts after exposure to hyperosmolar salt conditions
- The core protein to myogenesis NFAT5 forms aggresomes in normal and Duchenne muscular dystrophy myotubes exposed to cell stressors
- NFAT5 in inflammatory myopathies and Duchenne muscular dystrophy
- The core protein to myogenesis NFAT5 forms aggresomes in normal and Duchenne muscular dystrophy myotubes exposed to cell stressors
- 2015
- NFAT5 forms aggregates in normal and Duchenne muscular dystrophy cultured myotubes after exposure to cell stressors
- Inflammatory- and NaCl-induced expression of NFAT5 in muscle cells point to common stress response mechanisms
- Addition of NaCl upregulates NFAT5 expression in Duchenne muscular dystrophy cultured myotubes
- Activation of the nuclear factor of activated T-cells 5 pathway is characteristic of the perifascicular muscle fiber atrophy observed in dermatomyositis
- Pro-inflammatory cell stress in muscle cells is directed by NFAT5