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- Journal Article
- A1
- open access
Tumor endothelial cell autophagy is a key vascular-immune checkpoint in melanoma
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- Journal Article
- A1
- open access
NINJ1 is activated by cell swelling to regulate plasma membrane permeabilization during regulated necrosis
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- Journal Article
- A1
- open access
Cell death checkpoints in the TNF pathway
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- Journal Article
- A1
- open access
Inhibition of RIPK1 kinase does not affect diabetes development : beta-cells survive RIPK1 activation
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- Journal Article
- A1
- open access
From PERK to RIPK1 : design, synthesis and evaluation of novel potent and selective necroptosis inhibitors
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- Journal Article
- A1
- open access
LC3-independent autophagy is vital to prevent TNF cytotoxicity
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- Journal Article
- A1
- open access
Death by TNF : a road to inflammation
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Apoptotic cell death in disease : current understanding of the NCCD 2023
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- Journal Article
- A1
- open access
RIPK1 kinase-dependent inflammation and cell death contribute to the pathogenesis of COPD
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- Journal Article
- A1
- open access
ATG9A prevents TNF cytotoxicity by an unconventional lysosomal targeting pathway.
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- Journal Article
- A1
- open access
Phosphorylation of RIPK1 serine 25 mediates IKK dependent control of extrinsic cell death in T cells
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- Journal Article
- A1
- open access
A20 controls RANK-dependent osteoclast formation and bone physiology
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- Journal Article
- A1
- open access
Endocytic membrane repair by ESCRT-III controls antigen export to the cytosol during antigen cross-presentation
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- Journal Article
- A1
- open access
The Impact of RIPK1 Kinase Inhibition on Atherogenesis: A Genetic and a Pharmacological Approach
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- Journal Article
- A1
- open access
Antioxidant and food additive BHA prevents TNF cytotoxicity by acting as a direct RIPK1 inhibitor
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- Journal Article
- A1
- open access
Autophosphorylation at serine 166 regulates RIP kinase 1-mediated cell death and inflammation
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- Journal Article
- A1
- open access
Respiratory Syncytial Virus infection promotes necroptosis and HMGB1 release by airway epithelial cells
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RIPK1 kinase-dependent death : a symphony of phosphorylation events
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A20 and cell death-driven inflammation
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- Journal Article
- A1
- open access
Two distinct ubiquitin-binding motifs in A20 mediate its anti-inflammatory and cell-protective activities
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- Journal Article
- A1
- open access
OTULIN prevents liver inflammation and hepatocellular carcinoma by inhibiting FADD- and RIPK1 kinase-mediated hepatocyte apoptosis
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Immunodominant AH1 antigen-deficient necroptotic, but not apoptotic, murine cancer cells induce antitumor protection
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- Journal Article
- A1
- open access
Network approach identifies Pacer as an autophagy protein involved in ALS pathogenesis
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- Journal Article
- A1
- open access
A20 protects cells from TNF-induced apoptosis through linear ubiquitin-dependent and -independent mechanisms
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- Journal Article
- A1
- open access
Serine 25 phosphorylation inhibits RIPK1 kinase-dependent cell death in models of infection and inflammation
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Monitoring RIPK1 phosphorylation in the TNFR1 signaling complex
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MK2 puts an additional brake on RIPK1 cytotoxic potential
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The E3 ubiquitin ligases HOIP and cIAP1 are recruited to the TNFR2 signaling complex and mediate TNFR2-induced canonical NF-κB signaling
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Type III collagen affects dermal and vascular collagen fibrillogenesis and tissue integrity in a mutant Col3a1 transgenic mouse model
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- Journal Article
- A1
- open access
N-glycosylation of mouse TRAIL-R restrains TRAIL-induced apoptosis
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- Journal Article
- A1
- open access
Molecular mechanisms of cell death : recommendations of the Nomenclature Committee on Cell Death 2018
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MK2 phosphorylation of RIPK1 regulates TNF-mediated cell death
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- Journal Article
- A1
- open access
RIPK1 protects hepatocytes from death in Fas-induced hepatitis
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- Journal Article
- A1
- open access
Experimental African trypanosome infection suppresses the development of multiple myeloma in mice by inducing intrinsic apoptosis of malignant plasma cells
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When PERK inhibitors turn out to be new potent RIPK1 inhibitors : critical issues on the specificity and use of GSK2606414 and GSK2656157
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RIPK1 protects hepatocytes from Kupffer cells-mediated TNF-induced apoptosis in mouse models of PAMP-induced hepatitis
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Type III collagen is important for type I collagen fibrillogenesis and for dermal and cardiovascular development
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More to life than NF-κB in TNFR1 signaling
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Polyhydramnios, transient antenatal Bartter's syndrome, and MAGED2 mutations
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- Journal Article
- A1
- open access
Poly-ubiquitination in TNFR1-mediated necroptosis
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Differential expression of type III collagen in male and female mice
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Type III collagen is important for dermal and cardiovascular development, and type I collagen fibrillogenesis
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Type III collagen is important for type I collagen fibrillogenesis and for dermal and cardiovascular development
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- Journal Article
- A1
- open access
RIPK1 protects from TNF-α-mediated liver damage during hepatitis
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An evolutionary perspective on the necroptotic pathway
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A siRNA screen reveals the prosurvival effect of protein kinase A activation in conditions of unresolved endoplasmic reticulum stress
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A real-time fluorometric method for the simultaneous detection of cell death type and rate
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- Journal Article
- A1
- open access
The tumor suppressor Hace1 is a critical regulator of TNFR1-mediated cell fate
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- Journal Article
- A1
- open access
Vaccination with necroptotic cancer cells induces efficient anti-tumor immunity
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Regulation of RIPK1's cell death function by phosphorylation
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Type III collagen is important for type I collagen fibrillogenesis and for dermal and cardiovascular development
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- Journal Article
- A2
- open access
Molecular crosstalk between apoptosis, necroptosis, and survival signaling
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Type III collagen is important for collagen fibrillogenesis and for dermal and cardiovascular development
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cIAP2 supports viability of mice lacking cIAP1 and XIAP
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NIK promotes tissue destruction independently of the alternative NF-κB pathway through TNFR1/RIP1-induced apoptosis
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NF-κB-independent role of IKKα/IKKβ in preventing RIPK1 kinase-dependent apoptotic and necroptotic cell death during TNF signaling
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IAPs, regulators of innate immunity and inflammation
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- Journal Article
- A1
- open access
Endoplasmic reticulum stress induces ligand-independent TNFR1-mediated necroptosis in L929 cells
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- Journal Article
- A1
- open access
Essential versus accessory aspects of cell death: recommendations of the NCCD 2015
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Type III collagen is important for type I collagen fibrillogenesis and for dermal and cardiovascular development
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- Journal Article
- A1
- open access
RIPK1 promotes death receptor-independent caspase-8-mediated apoptosis under unresolved ER stress conditions
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Deficiency in the mitochondrial apoptotic pathway reveals the toxic potential of autophagy under ER stress conditions
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RIPK1 ensures intestinal homeostasis by protecting the epithelium against apoptosis
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- Journal Article
- A1
- open access
MLKL compromises plasma membrane integrity by binding to phosphatidylinositol phosphates
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- Journal Article
- A1
- open access
Depletion of RIPK3 or MLKL blocks TNF-driven necroptosis and switches towards a delayed RIPK1 kinase-dependent apoptosis
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Caspase-3 and RasGAP: a stress-sensing survival/demise switch
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C-propeptide mutations in procollagen type I and V are associated with endoplasmic reticulum stress-specific unfolded protein responses
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RIPK3 contributes to TNFR1-mediated RIPK1 kinase-dependent apoptosis in conditions of cIAP1/2 depletion or TAK1 kinase inhibition
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Loss of Maged1 results in obesity, deficits of social interactions, impaired sexual behavior and severe alteration of mature oxytocin production in the hypothalamus
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The role of the IAP E3 ubiquitin ligases in regulating pattern-recognition receptor signalling
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The unfolded protein response at the crossroads of cellular life and death during endoplasmic reticulum stress
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RIP1 is required for IAP inhibitor-mediated sensitization of childhood acute leukemia cells to chemotherapy-induced apoptosis
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Intermediate domain of receptor-interacting protein kinase 1 (RIPK1) determines switch between necroptosis and RIPK1 kinase-dependent apoptosis
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NOD-like receptors and the innate immune system: coping with danger, damage and death
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TNF-induced necroptosis in L929 cells is tightly regulated by multiple TNFR1 complex I and II members
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Smac mimetic bypasses apoptosis resistance in FADD- or caspase-8-deficient cells by priming for tumor necrosis factor α-induced necroptosis
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Interaction patches of procaspase-1 caspase recruitment domains (CARDs) are differently involved in procaspase-1 activation and receptor-interacting protein 2 (RIP2)-dependent nuclear factor κB signaling
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- Journal Article
- A1
- open access
cIAP1/2 are direct E3 ligases conjugating diverse types of ubiquitin chains to receptor interacting proteins kinases 1 to 4 (RIP1-4)
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The Ripoptosome: death decision in the Cytosol
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cIAP1 and TAK1 protect cells from TNF-induced necrosis by preventing RIP1/RIP3-dependent reactive oxygen species production
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ProNGF induces TNF alpha-dependent death of retinal ganglion cells through a p75(NTR) non-cell-autonomous signaling pathway
(2010) PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA. 107(8). p.3817-3822 -
- Journal Article
- A1
- open access
Maged1, a new regulator of skeletal myogenic differentiation and muscle regeneration
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RIP1's function in NF-kappa B activation: from master actor to onlooker
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Cellular inhibitors of apoptosis cIAP1 and cIAP2 are required for innate immunity signaling by the pattern recognition receptors NOD1 and NOD2
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- Journal Article
- A1
- open access
NRAGE, a p75NTR adaptor protein, is required for developmental apoptosis in vivo
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cIAP1 and cIAP2 facilitate cancer cell survival by functioning as E3 ligases that promote RIP1 ubiquitination
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Comparative expression analysis of the MAGED genes during embryogenesis and brain development
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An overview of the MAGE gene family with the identification of all human members of the family