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Specific Notch receptor-ligand interactions control human TCR-αβ/γδ development by inducing differential Notch signal strength
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Notch ligands control the human alpha beta/gamma delta lineage choice through receptor-specific induction of differential Notch signal strength
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Jagged2 acts as a Delta-like Notch ligand during early hematopoietic cell fate decisions
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An early decrease in Notch activation is required for human TCR-αβ lineage differentiation at the expense of TCR-γδ T cells
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Notch signaling is required for proliferation but not for differentiation at a well-defined β-selection checkpoint during human T-cell development
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Stage-dependent molecular changes in Notch signaling are critical for normal human T cell development
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Tumor necrosis factor promotes T-cell at the expense of B-cell lymphoid development from cultured human CD34(+) cord blood cells
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Notch signaling induces cytoplasmic CD3 epsilon expression in human differentiating NK cells
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Overexpression of HES-1 is not sufficient to impose T-cell differentiation on human hernatopoietic stem cells