Department of Biomedical molecular biology
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- 2015
- Essential versus accessory aspects of cell death: recommendations of the NCCD 2015
- TNFR1-induced lethal inflammation is mediated by goblet and Paneth cell dysfunction
- Leptin's metabolic and immune functions can be uncoupled at the ligand/receptor interaction level
- Endoplasmic reticulum stress induces ligand-independent TNFR1-mediated necroptosis in L929 cells
- A novel RIPK4-IRF6 connection is required to prevent epithelial fusions characteristic for popliteal pterygium syndromes